Free ARDMS AE-Adult-Echocardiography Practice Exam with Questions & Answers | Set: 2

The Valsalva maneuver decreases preload and left ventricular volume, which exacerbates left ventricular outflow tract obstruction in hypertrophic obstructive cardiomyopathy (HOCM). This results in an increase in the gradient and severity of obstruction and symptoms during the maneuver.

Aortic valve stenosis, aortic regurgitation, and mitral regurgitation typically decrease or do not significantly change during Valsalva because of decreased flow and pressure.

This physiological response is detailed in the "Textbook of Clinical Echocardiography, 6e", Chapter on Dynamic Left Ventricular Outflow Obstruction and Maneuvers【20:370-375†Textbook of Clinical Echocardiography】.

Comprehensive and Detailed Explanation From Exact Extract:

Cardiac tamponade occurs when fluid accumulation in the pericardial space increases intrapericardial pressure, restricting ventricular filling and reducing cardiac output. A hallmark clinical feature is hypotension due to decreased stroke volume and cardiac output.

Jugular venous pressure is typically elevated (not decreased) because of impaired right heart filling. Tachycardia, not bradycardia, is usually present as a compensatory response. Blood pressure tends to be low or normal, not hypertensive.

This pathophysiology and clinical presentation are well documented in adult echocardiography literature and clinical cardiology textbooks, where tamponade is diagnosed with signs such as right atrial and ventricular diastolic collapse and associated clinical hypotension and elevated venous pressures【16:Textbook of Clinical Echocardiography, 6e†p.280-285】【12:ASE Pericardial Disease Guidelines†p.300-305】.

Assessment of left ventricular diastolic function by echocardiography involves evaluating mitral inflow velocities with pulsed wave Doppler (E and A waves), pulmonary venous flow patterns (systolic and diastolic waves), and tissue Doppler imaging of the mitral annulus to measure early diastolic (e') velocities.

This combination allows differentiation of normal versus abnormal relaxation, elevated filling pressures, and grading of diastolic dysfunction. The myocardial performance index evaluates global ventricular function but does not specifically require these Doppler measures. Systolic function is assessed mainly by ejection fraction and wall motion. Mitral regurgitation severity uses color Doppler and vena contracta measurements.

This multiparameter diastolic function evaluation is outlined in the "Textbook of Clinical Echocardiography, 6e", Chapter on Diastolic Function Assessment【20:210-220†Textbook of Clinical Echocardiography】.

The image is a parasternal long-axis echocardiographic view focusing on the mitral valve annulus with a highly echogenic, dense, and well-defined structure located at the base of the posterior mitral leaflet. This appearance is characteristic of mitral annular calcification (MAC), a degenerative process resulting in calcium deposition along the mitral valve annulus.

Vegetations appear as irregular, mobile masses attached to valve leaflets and are less dense. Tumors and thrombi have different echogenicity and locations (tumors often in atria, thrombi in atrial appendages). MAC is usually more echogenic and localized to the annulus.

This description and differentiation are found in adult echocardiography textbooks and ASE guidelines on cardiac masses and valvular calcifications【16:Textbook of Clinical Echocardiography, 6e†p.460-465】【12:ASE Guidelines on Cardiac Masses†p.150-160】.

Comprehensive and Detailed Explanation From Exact Extract:

The P wave on the ECG corresponds to atrial depolarization, which precedes atrial contraction (atrial systole). On echocardiography, atrial contraction can be observed as the atrial "kick," contributing to ventricular filling during late diastole.

Ventricular contraction (QRS complex) and ventricular relaxation (T wave) correspond to other phases of the cardiac cycle. Atrial relaxation occurs during ventricular systole but is not represented by the P wave.

This timing relationship is critical for correlating echocardiographic Doppler inflow patterns, such as the late diastolic A wave, with the ECG. These concepts are outlined in the foundational echocardiography references, including ASE guidelines and the "Textbook of Clinical Echocardiography"【16:Textbook of Clinical Echocardiography, 6e†p.150-155】【12:ASE Echocardiography Guidelines†p.50-55】.

Comprehensive and Detailed Explanation From Exact Extract:

The Doppler waveform shows a typical left ventricular outflow tract or aortic valve velocity pattern. The arrow points to the steep rise and peak velocity of the jet, which corresponds to systolic ejection — the phase of the cardiac cycle when blood is rapidly ejected from the left ventricle into the aorta.

Isovolumic contraction precedes ejection and is represented by a flat baseline with no flow as ventricles build pressure. Isovolumic relaxation occurs after ejection before the mitral valve opens. Early diastole corresponds to mitral inflow, not aortic outflow.

This timing and flow pattern are standard in echocardiographic Doppler interpretation as described in the "Textbook of Clinical Echocardiography" and ASE Doppler imaging guidelines【16:Textbook of Clinical Echocardiography, 6e†p.100-105】【12:ASE Doppler Guidelines†p.50-55】.

Acoustic shadowing is the artifact caused by calcified structures like the mitral annulus, resulting in attenuation or loss of echoes from structures posterior to the calcification. The calcification absorbs or reflects the ultrasound waves, preventing them from reaching deeper structures and causing a "dropout" or dark shadow behind the calcified area.

Reverberation involves repeated reflections creating multiple echoes. Side lobe artifacts arise from off-axis beams. Ring-down artifacts result from resonance in fluid or gas bubbles, not calcifications.

This artifact is explained in the "Textbook of Clinical Echocardiography, 6e", Chapter on Ultrasound Artifacts【20:75-80†Textbook of Clinical Echocardiography】.QUESTION NO: 134

What is the range of the aortic valve area in normal adults?

A. 1 - 2 cm2

B. 3 - 4cm2

C. 5 - 6cm2

D. 7- 8cm2

Answer: B

Comprehensive and Detailed Explanation From Exact Extract:

The normal aortic valve area (AVA) in adults typically ranges from 3 to 4 cm². This measurement is important for assessing aortic stenosis severity; values below this range suggest valve narrowing.

AVA values of 1-2 cm² indicate mild to moderate stenosis, while less than 1 cm² reflects severe stenosis. Larger areas like 5-6 or 7-8 cm² are not physiologically typical.

This normal range is documented in the "Textbook of Clinical Echocardiography, 6e", Chapter on Aortic Valve Anatomy and Function【20:360-365†Textbook of Clinical Echocardiography】.

Decreasing the color scale (velocity range) improves the sensitivity of color Doppler for detecting low-velocity flow, such as shunting across the interatrial septum (e.g., patent foramen ovale). A lower scale allows subtle flow jets to be visualized.

Decreasing color gain would reduce sensitivity, increasing color sector size can degrade frame rate and resolution, and increasing the wall filter may remove low-velocity signals.

This optimization is discussed in the "Textbook of Clinical Echocardiography, 6e", Chapter on Color Doppler Imaging Techniques【20:100-105†Textbook of Clinical Echocardiography】.

Comprehensive and Detailed Explanation From Exact Extract:

A continuous murmur is a heart murmur that occurs throughout both systole and diastole. Among the options, a ruptured sinus of Valsalva aneurysm produces a continuous murmur due to persistent flow between the aorta and a cardiac chamber (usually the right atrium or ventricle) during both systole and diastole.

Aortic regurgitation causes a diastolic murmur, mitral stenosis causes a diastolic murmur, and a muscular ventricular septal defect typically causes a holosystolic murmur but not continuous.

Ruptured sinus of Valsalva aneurysm causes a continuous shunting of blood, resulting in the characteristic continuous murmur, often described as “machinery-like.”

This clinical correlation is covered in the "Textbook of Clinical Echocardiography, 6e", Chapter on Aortic Root and Sinus of Valsalva Pathology【20:420-425†Textbook of Clinical Echocardiography】.

Marfan syndrome is a connective tissue disorder characterized by abnormalities in the fibrillin-1 gene, leading to cardiovascular manifestations including aortic root and annular dilatation. Aortic annular dilatation predisposes to aortic valve insufficiency (regurgitation) and aortic aneurysm formation.

Coarctation of the aorta is more commonly associated with Turner syndrome. Parachute mitral valve and cleft mitral valve are congenital abnormalities linked to other syndromes or defects but not typical in Marfan syndrome.

This association is described in the "Textbook of Clinical Echocardiography, 6e", Chapter on Genetic Syndromes and Cardiovascular Manifestations【20:120-125†Textbook of Clinical Echocardiography】.QUESTION NO: 91

Which condition causes both tricuspid stenosis and tricuspid regurgitation?

A. Pulmonary hypertension

B. Cor pulmonale

C. Carcinoid heart disease

D. Amyloid heart disease

Answer: C

Comprehensive and Detailed Explanation From Exact Extract:

Carcinoid heart disease results from the deposition of fibrous plaques on the endocardium of right-sided heart valves, predominantly affecting the tricuspid and pulmonary valves. This leads to both tricuspid stenosis (valve leaflet thickening and immobility causing obstruction) and tricuspid regurgitation (incomplete coaptation due to leaflet retraction).

Pulmonary hypertension and cor pulmonale cause primarily functional tricuspid regurgitation without stenosis. Amyloid heart disease can cause restrictive cardiomyopathy but rarely causes combined tricuspid valve stenosis and regurgitation.

These pathological changes are detailed in the "Textbook of Clinical Echocardiography, 6e", Chapter on Carcinoid Heart Disease and Right Heart Valve Disease【20:335-340†Textbook of Clinical Echocardiography】.